Localized Hormone Manipulation for Penis Growth
Experimenting on using male hormone DHT transdermally on the penis to signal growth, let me explain how I understand the whole thing. First there is testosterone, which circulates in the blood and attaches and detaches from the androgen receptor sites in the body. Next, to control the level of testosterone, the body needs a way to destroy it. One of the possible ways is to use an alpha-reductase enzyme, which cleaves testosterone and the pieces it leaves behind is called dihydrotestosterone (dht). Dht is highly androgenic, which means that it attaches to the androgenic receptor sites longer and stronger. The penis contains a concentration of androgenic receptors, and if these receptors are saturated with dht hormone continuously, new cells (each containing more androgen receptors) are grown to help accommodate the local load of dht. (This is only speculation, as I am not an expert in this field.) However, if dht is taken over long periods of time, the body will shut down testosterone production in the testicles, since it believes that high dht levels mean that too much testosterone is being broken down by the alpha-reductase enzyme to dht.
Theories on the internet I tested was using dht gel and an alpha-reductase inhibitor called finasteride, which is a medication taken orally, but with a molecular weight that suggests a possibility for transdermal permeability with a dimethylsulfoxide carrier. The two are alternately taken, about a one for one then switching a month for the other. Finasteride protects the testosterone from being chopped up into dht, thus lowering overall dht. With low dht in the penis, the androgen receptors become more sensitive. This sensitivity translates to more efficient dht gel use, as each receptor activated sends a stronger and/or longer signal to the cell to produce growth. (Again, this is only theory on the exact mechanism of penile cell growth).
When I tried this, I did experience growth, but mainly girth. A few cycles in a row I stopped because the new thickness was becoming problematic for receiving blow jobs, and teeth became my bitter enemy. Penetrating anally was also an eye-opening experience for those on the far end of my dick, and the fear factor was rising.
But now, I have gotten used to the penile pain, and teeth were just a part of it. I want more size, goddammit! (-_-) So now I am repeating the experiment but with a different drug called dutosteride. It blocks the 2 types of alpha-reductase enzyme, while finasteride blocks only one type, therefore, it should be more efficient in reducing dht. It is a gelcap taken orally.
One of the symptoms of low dht is reduced libido, which is something of a bummer. But remember, the mind is quite strong and mental fantasy can bring back the mood; its just a little more challenging. At least the erection injections help too! The dht phase is the opposite, where horniness is at its maximum. The problem then is physical over-use and chaffing. Use more lube for this problem to reduce friction.
Thanks for reading my blog!
Theories on the internet I tested was using dht gel and an alpha-reductase inhibitor called finasteride, which is a medication taken orally, but with a molecular weight that suggests a possibility for transdermal permeability with a dimethylsulfoxide carrier. The two are alternately taken, about a one for one then switching a month for the other. Finasteride protects the testosterone from being chopped up into dht, thus lowering overall dht. With low dht in the penis, the androgen receptors become more sensitive. This sensitivity translates to more efficient dht gel use, as each receptor activated sends a stronger and/or longer signal to the cell to produce growth. (Again, this is only theory on the exact mechanism of penile cell growth).
When I tried this, I did experience growth, but mainly girth. A few cycles in a row I stopped because the new thickness was becoming problematic for receiving blow jobs, and teeth became my bitter enemy. Penetrating anally was also an eye-opening experience for those on the far end of my dick, and the fear factor was rising.
But now, I have gotten used to the penile pain, and teeth were just a part of it. I want more size, goddammit! (-_-) So now I am repeating the experiment but with a different drug called dutosteride. It blocks the 2 types of alpha-reductase enzyme, while finasteride blocks only one type, therefore, it should be more efficient in reducing dht. It is a gelcap taken orally.
One of the symptoms of low dht is reduced libido, which is something of a bummer. But remember, the mind is quite strong and mental fantasy can bring back the mood; its just a little more challenging. At least the erection injections help too! The dht phase is the opposite, where horniness is at its maximum. The problem then is physical over-use and chaffing. Use more lube for this problem to reduce friction.
Thanks for reading my blog!